11/22/2023 0 Comments Chronic sleep insomnia![]() The lack of sleep associated with insomnia can happen for a number of reasons, including stress, work schedule, poor sleeping habits, late-night eating, caffeine, nicotine, and alcohol. Our physician-owned practice is dedicated to your overall health through custom treatment plans and lifestyle changes to help you sleep better. Sarah Patel and Vinod Patel and the experienced team at Sonoran Sleep Center. Residents of the Glendale, Arizona area who are looking for relief from sleep disorders can find help with Drs. To better understand the damage insomnia can do, let’s look at what causes this condition, the different types you can find yourself dealing with, and the dangers it poses to your health. Chronic insomnia can seriously affect your body mentally and physically. The American Academy of Sleep Medicine estimates that up to 35% of people deal with brief insomnia, up to 20% deal with insomnia for less than three months, and 10% deal with chronic insomnia, which can happen three times a week for at least three months. ![]() One of the most common forms of sleep disorder is insomnia, a problem that ranges from an occasional nuisance to a chronic condition that increases the risks of even worse conditions. Even mild issues can affect your productivity and stress levels. Unfortunately, millions of people struggle with sleeping problems and the resulting effects. Sustained attention was not affected.Sleeping isn’t just something you do when you get tired at some point in the evening, it’s an essential function of the body for your physical and mental health. The results show that melatonin, 5 mg at 6 PM, was relatively safe to take in the short term and significantly more effective than placebo in advancing sleep onset and dim light melatonin onset and increasing sleep duration in elementary school children with chronic sleep onset insomnia. One child developed mild generalized epilepsy 4 months after the start of the trial. Twelve children used melatonin 5 mg, the other 1.0 to 2.5 mg. Eighteen months after the start of the trial, in 13 of the 38 children who could be followed up, melatonin treatment was stopped because their sleep problem was solved and in 1 child because sleep was not improved. Mild headache occurred in 2 children during the first 2 days of the melatonin treatment. There were no significant differences between the treatment groups in the change of sleep latency, wake-up time, and sustained attention reaction times. The change during the 4-week treatment period differed between the treatment groups significantly as to lights-off time, diary and actigraphic sleep onset, sleep duration, and melatonin onset. In the placebo group, these parameters did not shift significantly. In the melatonin group, mean (95% CI) lights-off time advanced 34 (6-63) minutes, diary sleep onset 63 (32-94) minutes, actigraphic sleep onset 75 (36-114) minutes, and melatonin onset 57 (24 to 89) minutes total sleep time increased 41 (19-62) minutes. Sustained attention was evaluated with the Bourdon-Vos reaction time test (n = 36). Endogenous dim light melatonin onset was measured in saliva (n = 27). Sleep onset was also recorded with an actigraph (n = 25). The study's impact was assessed by measurements of lights-off time, sleep onset, and wake-up time, recorded in a diary (n = 33). After that period, treatment was continued if the parents wished so. The study consisted of a 1-week baseline, consecutively followed by a 4-week treatment period. The children were randomly assigned to receive either 5-mg melatonin or placebo. To establish the efficacy of melatonin treatment in childhood sleep onset insomnia, 40 elementary school children, 6 to 12 years of age, who suffered more than 1 year from chronic sleep onset insomnia, were studied in a double-blind, placebo-controlled study.
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